
PBPs are more than static β lactam targets. Host conditions rewire PBP activity and peptidoglycan architecture, shaping tolerance, resistance, and how we design salvage regimens.
Black is an NIH/NCATS T32 postdoctoral research fellow at UT Health San Antonio and an adjoint assistant professor at The University of Texas at Austin College of Pharmacy. A clinical staff pharmacist at University Hospital (San Antonio), his work centers on antimicrobial resistance, β-lactam pharmacology, and translational infectious diseases research. (ORCID: 0000-0003-0020-0216)

PBPs are more than static β lactam targets. Host conditions rewire PBP activity and peptidoglycan architecture, shaping tolerance, resistance, and how we design salvage regimens.